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Hence, by targeting the enzymatic activity of SARM1, inhibitors may effectively prevent the depletion of NAD + , thereby maintaining cellular energy homeostasis and enhancing neuronal resilience, potentially expanding the treatment landscape for various neurological conditions

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This observation was more pronounced when gallbladder adenocarcinoma was present when compared to gallbladder dysplasia: HR 2.25

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Navaneetharaja N, Griffiths V, Wileman T, Carding SR

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